![]() There was compelling evidence that cognitive dysfunction is associated with an aberrant functional brain network (Maher et al., 2015 Mikell et al., 2015 Nelson et al., 2016). Numerous neuroimaging studies have been performed to explore cognitive deficits caused by neurological problems, including hemorrhagic cerebrovascular disease (Baldassarre et al., 2016 Siegel et al., 2016 Adhikari et al., 2017 Chung et al., 2021). However, the neural mechanisms underlying cognitive deficits after the rupture of ACoA aneurysm remain unclear, resulting in limited treatment. Therefore, there is an urgent clinical need for the treatment of cognitive dysfunction following the rupture of cerebral aneurysm. Cognitive impairment reportedly occurs in approximately half of the surviving patients, of which memory loss is the most common complaint, preventing them from returning to their normal life (Beeckmans et al., 2020 Neifert et al., 2021). ![]() Subarachnoid hemorrhage (SAH) caused by the rupture of anterior communicating artery (ACoA) aneurysm is a devastating cerebrovascular event with high morbidity and mortality (Hong et al., 2012 Ito et al., 2015).
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